Sun Pharmaceutical Industries Ltd
Salt CompositionEdaravone (1.5mg)
StorageStore below 30°C
(Inclusive of all taxes)
Pack Size20 ml in 1 vial
View alternate brandAravon Injection is used in the treatment of amyotrophic lateral sclerosis (a nervous system disease that weakens the muscles and impairs physical functions). It is an antioxidant that slows down nerve damage and prevents further progression of the disease.
Aravon Injection is administered as an injection under the supervision of a healthcare professional and should not be self-administered. It is given in cases where oral administration is not possible. Take this medicine in the dose and duration as advised by your doctor and if you have missed a dose, take it as soon as you remember.
The most common side effects of this medicine are headache, bruising, and walking difficulties (gait disturbance). You may also notice some injection site reactions such as pain, redness, or swelling. However, these are temporary and usually resolve on their own. Please consult your doctor if these do not subside or bother you. You should inform the doctor if you develop any allergic reactions like itching, swelling of the lips, tongue, or face, after taking the medication as these require immediate medical attention.
Amyotrophic lateral sclerosis (ALS) is a disease of the nerves that restricts muscle movements and hence affects the physical functions of the affected person. Aravon Injection improves nerve activity and muscle strength. This helps the person to carry out the daily activities like walking, talking, chewing, etc, more conveniently. Hence, the person becomes more confident.
Most side effects do not require any medical attention and disappear as your body adjusts to the medicine. Consult your doctor if they persist or if you’re worried about them
Your doctor or nurse will give you this medicine. Kindly do not self administer.
Aravon Injection is an antioxidant. It has nootropic (enrichment of nerves) and neuroprotective properties. It works by slowing the nerve damage caused by harmful chemicals (free radicals). This delays the progression of amyotrophic lateral sclerosis (ALS) symptoms by reducing the oxidative stress (an imbalance between free radicals and antioxidants) in the brain.
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